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1.
Koob, D.* et al.: Copper accumulation in chronic cholestatic disease augments liver damage by impairment of mitochondrial function. J. Hepatol. 77, S602-S603 (2022)
2.
Sandforth, L. ; El-Agroudy, N.N. & Birkenfeld, A.L.: Is NAFLD a key driver of brain dysfunction? J. Hepatol., DOI: 10.1016/j.jhep.2022.09.022 (2022)
3.
Quitt, O. et al.: T-cell engager antibodies enable T cells to control HBV infection and to target HBsAg-positive hepatoma in mice. J. Hepatol. 75, 1058-1071 (2021)
4.
Teng, Y.* et al.: Novel function of SART1 in HNF4α transcriptional regulation contributes to its antiviral role during HBV infection. J. Hepatol. 75, 1072-1082 (2021)
5.
Wing, P.A.* et al.: Hypoxia inducible factors regulate hepatitis B virus replication by activating the basal core promoter. J. Hepatol. 75, 64-73 (2021)
6.
Yuan, S.* et al.: Translatomic profiling reveals novel self-restricting virus-host interactions during HBV infection. J. Hepatol. 75, 74-85 (2021)
7.
Lampl, S.* et al.: Reduced mitochondrial resilience enables non-canonical induction of apoptosis after TNF receptor signaling in virus-infected hepatocytes. J. Hepatol. 73, 1347-1359 (2020)
8.
Lucifora, J.* et al.: Two-dimensional-cultures of primary human hepatocytes allow efficient HBV infection: Old tricks still work! J. Hepatol. 73, 449-451 (2020)
9.
Namineni, S. et al.: A dual role for hepatocyte-intrinsic canonical NF-κB signaling in virus control. J. Hepatol. 72, 960-975 (2020)
10.
Pietrocola, F.* ; Castoldi, F.* ; Zischka, H. & Kroemer, G.*: Extending the mode of action of triethylenetetramine (trientine): Autophagy besides copper chelation. J. Hepatol. 73, 970-972 (2020)
11.
Schwerbel, K.* et al.: Immunity-related GTPase induces lipophagy to prevent excess hepatic lipid accumulation. J. Hepatol. 73, 771-782 (2020)
12.
Marbach-Breitrück, E.* et al.: Tick-tock hedgehog-mutual crosstalk with liver circadian clock promotes liver steatosis. J. Hepatol. 70, 1192-1202 (2019)
13.
Oberhardt, V.* et al.: Viral escape contributes to the failure of hepatitis D virus-specific CD8+T-cells and drives evolution of HDV. J. Hepatol. 70, E21-E21 (2019)
14.
Schreiber, S. et al.: Characterization of a library of CD4+T cells for T-cell therapy of HBV infection. J. Hepatol. 70, E456-E456 (2019)
15.
Su, J. et al.: Exploring TH1/TH2 adjuvants to improve the efficacy of the therapeutic vaccination against chronic hepatitis B. J. Hepatol. 70, E485-E486 (2019)
16.
Velkov, S. ; Protzer, U. & Michler, T.: Approximation of the genotype distribution within global chronic hepatitis B virus infections. J. Hepatol. 70, E711-E711 (2019)
17.
Wilman, H.R.* et al.: Genetic studies of abdominal MRI data identify genes regulating hepcidin as major determinants of liver iron concentration. J. Hepatol. 71, 594-602 (2019)
18.
Einer, C. ; Leitzinger, C. ; Hohenester, S.* ; Weiss, K.H.* & Zischka, H.: High caloric nutrition promotes hepatocellular damage and steatohepatitis in Wilson disease rats via amplified mitochondrial damage. J. Hepatol. 68, S626-S626 (2018)
19.
Ko, C. et al.: Hepatitis B virus genome recycling and de novo secondary infection events maintain stable cccDNA levels. J. Hepatol. 69, 1231-1241 (2018)
20.
Michler, T. et al.: Combinatorial RNAi/vaccination therapy for chronic hepatitis B achieves long-term functional cure in preclinical mouse model. J. Hepatol. 68, S16-S16 (2018)