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Distler, M.* ; Aust, D.* ; Weitz, J.* ; Pilarsky, C.* ; Grützmann, R.*

Precursor lesions for sporadic pancreatic cancer: PanIN, IPMN, and MCN.

Biomed Res. Int. 2014:474905 (2014)
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Open Access Gold möglich sobald Verlagsversion bei der ZB eingereicht worden ist.
Pancreatic cancer is still a dismal disease. The high mortality rate is mainly caused by the lack of highly sensitive and specific diagnostic tools, and most of the patients are diagnosed in an advanced and incurable stage. Knowledge about precursor lesions for pancreatic cancer has grown significantly over the last decade, and nowadays we know that mainly three lesions (PanIN, and IPMN, MCN) are responsible for the development of pancreatic cancer. The early detection of these lesions is still challenging but provides the chance to cure patients before they might get an invasive pancreatic carcinoma. This paper focuses on PanIN, IPMN, and MCN lesions and reviews the current level of knowledge and clinical measures.
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Publikationstyp Artikel: Journalartikel
Dokumenttyp Wissenschaftlicher Artikel
ISSN (print) / ISBN 2314-6133
e-ISSN 2314-6141
Zeitschrift BioMed Research International
Quellenangaben Band: 2014, Heft: , Seiten: , Artikelnummer: 474905 Supplement: ,
Verlag Hindawi
Verlagsort [New York, NY] [u.a.]
Begutachtungsstatus
Institut(e) Institute for Pancreatic Beta Cell Research (IPI)