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1.
Braenne, I.* et al.: Genomic correlates of glatiramer acetate adverse cardiovascular effects lead to a novel locus mediating coronary risk. PLoS ONE 12:e0182999 (2017)
2.
Braenne, I.* et al.: A genomic exploration identifies mechanisms that may explain adverse cardiovascular effects of COX-2 inhibitors. Sci. Rep. 7:10252 (2017)
3.
Eitel, I.* et al.: Genome-wide association study in takotsubo syndrome - Preliminary results and future directions. Int. J. Cardiol. 236, 335-339 (2017)
4.
Howson, J.M.M.* et al.: Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms. Nat. Genet. 49, 1113-1119 (2017)
5.
Saleheen, D.* et al.: Loss of cardioprotective effects at the ADAMTS7 locus as a result of gene-smoking interactions. Circulation 135, 2336-2353 (2017)
6.
Ventura-Clapier, R.* et al.: Sex in basic research - Concepts in the cardiovascular field. Cardiovasc. Res. 113, 711-724 (2017)
7.
Webb, T.R.* et al.: Systematic evaluation of pleiotropy identifies 6 further loci associated with coronary artery disease. J. Am. Coll. Cardiol. 69, 823-836 (2017)
8.
Aherrahrou, Z* et al.: Knock-out of nexilin in mice leads to dilated cardiomyopathy and endomyocardial fibroelastosis. Basic Res. Cardiol. 111:6 (2016)
9.
Braenne, I.* et al.: Pleiotropic molecular targets of coxibs reveals novel genomic loci conferring coronary artery disease risk. Atherosclerosis 252, E252-E253 (2016)
10.
Ehret, G.B.* et al.: The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals. Nat. Genet. 48, 1171-1184 (2016)
11.
Golbus, J.R.* et al.: Common and rare genetic variation in CCR2, CCR5, or CX3CR1 and risk of atherosclerotic coronary heart disease and glucometabolic traits. Circ. Cardiovasc. Genet. 9, 250-258 (2016)
12.
Meindl, C.* et al.: Coronary artery ectasia are frequently observed in patients with bicuspid aortic valves with and without dilatation of the ascending aorta. Circ. Cardiovasc. Intervent. 9:e004092 (2016)
13.
Scott, R.A.* et al.: A genomic approach to therapeutic target validation identifies a glucose-lowering GLP1R variant protective for coronary heart disease. Sci. Transl. Med. 8:341ra76 (2016)
14.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N. Engl. J. Med. 374, 1134-1144 (2016)
15.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease (vol 374, pg 1134, 2016). N. Engl. J. Med. 374, 1898-1898 (2016)
16.
van der Laan, S.W.* et al.: Cystatin C and cardiovascular disease: A mendelian randomization study. J. Am. Coll. Cardiol. 68, 934-945 (2016)
17.
Winkler, T.W.* et al.: Correction: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 12:e1006166 (2016)
18.
Zanoni, P.* et al.: Rare variant in scavenger receptor BI raises HDL cholesterol and increases risk of coronary heart disease. Science 351, 1166-1671 (2016)
19.
Interleukin 1 Genetics Consortium et al.: Cardiometabolic effects of genetic upregulation of the interleukin 1 receptor antagonist: A mendelian randomisation analysis. Lancet Diabet. Endocrinol. 3, 243-253 (2015)
20.
Asl, H.F.* et al.: Expression quantitative trait loci acting across multiple tissues are enriched in inherited risk for coronary artery disease. Circ. Cardiovasc. Genet. 8, 305-315 (2015)