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9 Records found.
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1.
Schludi, M.H.* et al.: Spinal poly-GA inclusions in a C9orf72 mouse model trigger motor deficits and inflammation without neuron loss. Acta Neuropathol. 134, 241–254 (2017)
2.
Zhou, Q.* et al.: Antibodies inhibit transmission and aggregation of C9orf72 poly-GA dipeptide repeat proteins. EMBO Mol. Med. 9, 687-702 (2017)
3.
Schludi, M.H.* et al.: Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing. Acta Neuropathol. 130, 537-555 (2015)
4.
Schludi, M.H.* et al.: Erratum to: Distribution of dipeptide repeat proteins in cellular models and C9orf72 mutation cases suggests link to transcriptional silencing. Acta Neuropathol. 130, 557-558 (2015)
5.
Arzberger, T.* et al.: Neuropathological alterations in ftd cases with c9orf72 mutation - new insights. Eur. Psych. 29:EPA-1718 (2014)
6.
Brockmann, A.* et al.: Structure-function relationship of thiazolide-induced apoptosis in colorectal tumor cells. ACS Chem. Biol. 9, 1520-1527 (2014)
7.
May, S.* et al.: C9orf72 FTLD/ALS-associated Gly-Ala dipeptide repeat proteins cause neuronal toxicity and Unc119 sequestration. Acta Neuropathol. 128, 485-503 (2014)
8.
Mori, K.* et al.: The C9orf72 GGGGCC repeat is translated into aggregating dipeptide-repeat proteins in FTLD/ALS. Science 339, 1335-1338 (2013)
9.
Mori, K.* et al.: Bidirectional transcripts of the expanded C9orf72 hexanucleotide repeat are translated into aggregating dipeptide repeat proteins. Acta Neuropathol. 126, 881-893 (2013)