PuSH - Publication Server of Helmholtz Zentrum München

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1.
Moore, C.* et al.: Resequencing study confirms host defense and cell senescence gene variants contribute to the risk of idiopathic pulmonary fibrosis. Am. J. Respir. Crit. Care Med., accepted (2019)
2.
Sakornsakolpat, P.* et al.: Genetic landscape of chronic obstructive pulmonary disease identifies heterogeneous cell-type and phenotype associations. Nat. Genet. 51, 494-505 (2019)
3.
Shrine, N.* et al.: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries. Nat. Genet. 51, 481-493 (2019)
4.
Shrine, N.* et al.: Author Correction: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries (Nature Genetics, (2019), 51, 3, (481-493), 10.1038/s41588-018-0321-7). Nat. Genet., accepted (2019)
5.
Wain, L.V.* et al.: Genome-wide association analyses for lung function and chronic obstructive pulmonary disease identify new loci and potential druggable targets. Nat. Genet. 49, 416-425 (2017)
6.
Silverman, E.K.* et al.: Opportunities and challenges in the genetics of COPD 2010: An International COPD Genetics Conference report. COPD-J. Chronic Obstr. Pulm. Dis. 8, 121-135 (2011)
7.
Stahlhofen, W.: Human Lung Clearance Following Bolus Inhalation of Radioaerosols. In: Crapo, J.D.* [Eds.]: Extrapolation of Dosimetric Relationships for Inhaled Particles and Gases. New York : Academic Press, 1989. 153-166