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1.
Justice, A.E.* et al.: Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution. Nat. Genet. 51, 452–469 (2019)
2.
Turcot, V.* et al.: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity. Nat. Genet. 50, 26-41 (2018)
3.
Turcot, V.* et al.: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity (vol 50, pg 765, 2017). Nat. Genet. 50, 764-768 (2018)
4.
Holzinger, E.R.* et al.: Discovery and replication of SNP-SNP interactions for quantitative lipid traits in over 60,000 individuals. BioData Min. 10:25 (2017)
5.
Holmes, M.V.* et al.: Mendelian randomization of blood lipids for coronary heart disease. Eur. Heart J. 36, 539-550 (2015)
6.
Asselbergs, F.W.* et al.: Large-scale gene-centric meta-analysis across 32 studies identifies multiple lipid loci. Am. J. Hum. Genet. 91, 823-838 (2012)
7.
Morris, A.P.* et al.: Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nat. Genet. 44, 981-990 (2012)
8.
Lanktree, M.B.* et al.: Meta-analysis of dense genecentric association studies reveals common and uncommon variants associated with height. Am. J. Hum. Genet. 88, 6-18 (2011)
9.
Schunkert, H.* et al.: Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease. Nat. Genet. 43, 333-340 (2011)
10.
Sarwar, N.* et al.: Triglyceride-mediated pathways and coronary disease: Collaborative analysis of 101 studies. Lancet 375, 1634-1639 (2010)