PuSH - Publication Server of Helmholtz Zentrum München

23 Records found.
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1.
Justice, A.E.* et al.: Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution. Nat. Genet. 51, 452–469 (2019)
2.
Kunkle, B.W.* et al.: Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing. Nat. Genet. 51, 414-430 (2019)
3.
Evangelou, E.* et al.: Genetic analysis of over 1 million people identifies 535 new loci associated with blood pressure traits. Nat. Genet. 50, 1412-1425 (2018)
4.
Evangelou, E.* et al.: Erratum to: Genetic analysis of over 1 million people identifies 535 new loci associated with blood pressure traits (Nature Genetics, (2018), 50, 10, (1412-1425), 10.1038/s41588-018-0205-x). Nat. Genet., accepted (2018)
5.
Flannick, J.* et al.: Sequence data and association statistics from 12,940 type 2 diabetes cases and controls. Sci. Data 5:180002 (2018)
6.
Ligthart, S.* et al.: Genome analyses of >200,000 individuals identify 58 loci for chronic inflammation and highlight pathways that link inflammation and complex disorders. Am. J. Hum. Genet. 103, 691-706 (2018)
7.
Mahajan, A.* et al.: Refining the accuracy of validated target identification through coding variant fine-mapping in type 2 diabetes. Nat. Genet. 50, 559-571 (2018)
8.
Mahajan, A.* et al.: Fine-mapping type 2 diabetes loci to single-variant resolution using high-density imputation and islet-specific epigenome maps. Nat. Genet. 50, 1505-1513 (2018)
9.
Flannick, J.* et al.: Sequence data and association statistics from 12,940 type 2 diabetes cases and controls. Sci. Data 4:170179 (2017)
10.
Nolte, I.M.* et al.: Genetic loci associated with heart rate variability and their effects on cardiac disease risk. Nat. Commun. 8:15805 (2017)
11.
Wain, L.V.* et al.: Novel blood pressure locus and gene discovery using genome-wide association study and expression data sets from blood and the kidney. Hypertension 70, e4-e19 (2017)
12.
Fall, T.* et al.: Non-targeted metabolomics combined with genetic analyses identifies bile acid synthesis and phospholipid metabolism as being associated with incident type 2 diabetes. Diabetologia 59, 2114-2124 (2016)
13.
Fuchsberger, C.* et al.: The genetic architecture of type 2 diabetes. Nature 536, 41-47 (2016)
14.
Nowak, C.* et al.: Effect of insulin resistance on monounsaturated fatty acid levels: A multi-cohort non-targeted metabolomics and mendelian randomization study. PLoS Genet. 12:e1006379 (2016)
15.
van der Laan, S.W.* et al.: Cystatin C and cardiovascular disease: A mendelian randomization study. J. Am. Coll. Cardiol. 68, 934-945 (2016)
16.
Fall, T.* et al.: Age- and sex-specific causal effects of adiposity on cardiovascular risk factors. Diabetes 64, 1841-1852 (2015)
17.
Hägg, S.* et al.: Adiposity as a cause of cardiovascular disease: A Mendelian randomization study. Int. J. Epidemiol. 44, 578-586 (2015)
18.
Locke, A.E.* et al.: Genetic studies of body mass index yield new insights for obesity biology. Nature 518, 197-206 (2015)
19.
Shungin, D.* et al.: New genetic loci link adipose and insulin biology to body fat distribution. Nature 518, 187-196 (2015)
20.
Willem, M.* et al.: η-secretase processing of APP inhibits neuronal activity in the hippocampus. Nature 526, 443-447 (2015)