PuSH - Publication Server of Helmholtz Zentrum München

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1.
Thomas, J. et al.: Is the humoral immunity dispensable for the pathogenesis of psoriasis? J. Eur. Acad. Dermatol. Venereol. 33, 115-122 (2019)
2.
Thomas, J. et al.: Foxo4 and AHR control a stress-induced immune-mediated host protection via secretion of IL-22. Exp. Dermatol. 28, E76-E77 (2019)
3.
Garzorz-Stark, N.* et al.: Toll-like receptor 7/8 agonists stimulate plasmacytoid dendritic cells to initiate T(H)17-deviated acute contact dermatitis in human subjects. J. Allergy Clin. Immunol. 141, 1320-1333.e11 (2018)
4.
Thomas, J. et al.: The role of humoral immunity in the pathogenesis of psoriasis. Allergy 73, 732-733 (2018)
5.
Garzorz-Stark, N. et al.: The imiquimod model as a model for psoriasis and/or eczema in humans? Exp. Dermatol. 26, E68-E68 (2017)
6.
Mattii, M. et al.: Sebocytes contribute to skin inflammation by promoting the differentiation of T helper 17 cells. Br. J. Dermatol. 178, 722-730 (2017)
7.
Garzorz-Stark, N.* et al.: Pyoderma gangrenosum, Acne, Psoriasis, Arthritis and Suppurative Hidradenitis (PAPASH)-syndrome: A new entity within the spectrum of autoinflammatory syndromes? J. Eur. Acad. Dermatol. Venereol. 30, 141–143 (2016)
8.
Garzorz-Stark, N.* et al.: A novel molecular disease classifier for psoriasis and eczema. Exp. Dermatol. 25, 767-774 (2016)
9.
Garzorz, N.V.* et al.: Molecular diagnostics of psoriasis and eczema-a novel approach to establish personalised therapy. J. Invest. Dermatol. 135, S1 (2015)
10.
Garzorz-Stark, N.* et al.: Dissecting susceptibility from exogenous triggers: The model of Alopecia areata and associated inflammatory skin diseases. J. Eur. Acad. Dermatol. Venereol. 29, 2429–2435 (2015)