PuSH - Publication Server of Helmholtz Zentrum München

19 Records found.
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1.
Brazel, D.M.* et al.: Exome chip meta-analysis fine maps causal variants and elucidates the genetic architecture of rare coding variants in smoking and alcohol use. Biol. Psychiatry, accepted (2019)
2.
Erzurumluoglu, A.M.* et al.: Meta-analysis of up to 622,409 individuals identifies 40 novel smoking behaviour associated genetic loci. Mol. Psychiatry, accepted (2019)
3.
Jiang, X.* et al.: Shared heritability and functional enrichment across six solid cancers. Nat. Commun. 10:431 (2019)
4.
Li, Y.* et al.: Genetic interaction analysis among oncogenesis-related genes revealed novel genes and networks in lung cancer development. Oncotarget 10, 1760-1774 (2019)
5.
Zhu, Y.* et al.: Elevated platelet count appears to be causally associated with increased risk of lung cancer: A Mendelian randomization analysis. Cancer Epidemiol. Biomarkers Prev. 28, 935-942 (2019)
6.
Brokhman, I.* et al.: Dual embryonic origin of the mammalian enteric nervous system. Dev. Biol., accepted (2018)
7.
Ferreiro-Iglesias, A.* et al.: Fine mapping of MHC region in lung cancer highlights independent susceptibility loci by ethnicity. Nat. Commun. 9:3927 (2018)
8.
Ji, X.* et al.: Identification of susceptibility pathways for the role of chromosome 15q25.1 in modifying lung cancer risk. Nat. Commun. 9:3221 (2018)
9.
Kachuri, L.* et al.: Mendelian Randomization and mediation analysis of leukocyte telomere length and risk of lung and head and neck cancers. Int. J. Epidemiol., accepted (2018)
10.
Li, Y.* et al.: Genome-wide interaction study of smoking behavior and non-small cell lung cancer risk in Caucasian population. Carcinogenesis 39, 336-346 (2018)
11.
Rosenberger, A.* et al.: Genetic modifiers of radon-induced lung cancer risk: A genome-wide interaction study in former uranium miners. Int. Arch. Occup. Environ. Health 91, 937-950 (2018)
12.
Théry, C.* et al.: Minimal information for studies of extracellular vesicles 2018 (MISEV2018): A position statement of the International Society for Extracellular Vesicles and update of the MISEV2014 guidelines. J. Extra. Vesicles 7, 1535750 (2018)
13.
Zhang, W.* et al.: Different pathogen defense strategies in Arabidopsis: More than pathogen recognition. Cells 7:252 (2018)
14.
Carreras-Torres, R.* et al.: Obesity, metabolic factors and risk of different histological types of lung cancer: A Mendelian randomization study. PLoS ONE 12:e0177875 (2017)
15.
Ezzati, M.* et al.: Worldwide trends in body-mass index, underweight, overweight, and obesity from 1975 to 2016: A pooled analysis of 2416 population-based measurement studies in 128.9 million children, adolescents, and adults. Lancet 390, 2627-2642 (2017)
16.
McKay, J.D.* et al.: Large-scale association analysis identifies new lung cancer susceptibility loci and heterogeneity in genetic susceptibility across histological subtypes. Nat. Genet. 49, 1126-1132 (2017)
17.
Zhou, F.S.* et al.: Susceptibility loci of CNOT6 in the general mRNA degradation pathway and lung cancer risk - a re-analysis of eight GWASs. Mol. Carcinog. 56, 1227-1238 (2017)
18.
NCD Risk Factors Collaboration et al.: Trends in adult body-mass index in 200 countries from 1975 to 2014: A pooled analysis of 1698 population-based measurement studies with 19.2 million participants. Lancet 387, 1377-1396 (2016)
19.
Brenner, D.R.* et al.: Erratum to: Hierarchical modeling identifies novel lung cancer susceptibility variants in inflammation pathways among 10,140 cases and 11,012 controls. Hum. Genet. 135, 963-963 (2016)