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1.
Carlet, M. et al.: Inhibitor of apoptosis proteins represent attractive therapeutic target for relapsed/refractory ALL. Poster: 6th Munich Cancer Retreat, 3-4 July 2019, Munich, Germany. (2019)
2.
Carlet, M.* et al.: Smac mimetics induces synergistic apoptosis when combined with cytotoxic drugs, independently from TNFalpha and NFkappaB, but similarly to silencing of XIAP in ALL . Poster: Wildsede Meeting 2018, 30 June - 03 July 2018, Wildsede. (2018)
3.
Carlet, M. et al.: Synergistic apoptosis induced by the combination of Smac mimetics and cytotoxic drugs, occurs independently from TNFalpha and NFkappaB, but similarly to inhibition of XIAP in ALL. Poster: Frankfurt Cancer Conference, 25-27 September 2018, Frankfurt. (2018)
4.
Renner, S.* et al.: Metabolic syndrome and extensive adipose tissue inflammation in morbidly obese Göttingen minipigs. Mol. Metab., accepted (2018)
5.
Vergalli, J. et al.: XIAP is an attractive therapeutic target for all as validated in innovative genetically rngineered patient-derived xenograft models. Ann. Hematol. 96, S74-S75 (2017)
6.
Vergalli, J. et al.: Novel genetically engineered patient-derived xenograft (GEPDX) models reveal that xiap plays an essential role for patients´all growing in mice. Exp. Hematol. 44, S105-S105 (2016)