PuSH - Publication Server of Helmholtz Zentrum München

16 Records found.
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1.
Weber, J.* et al.: PiggyBac transposon tools for recessive screening identify B-cell lymphoma drivers in mice. Nat. Commun. 10:1415 (2019)
2.
Mueller, S.* et al.: Evolutionary routes and KRAS dosage define pancreatic cancer phenotypes. Nature 554, 62-68 (2018)
3.
Mueller, S.* et al.: Evolutionary trajectories and KRAS gene dosage define pancreatic cancer phenotypes. Cancer Res. 78 (2018)
4.
Tzelepis, K.* et al.: SRPK1 maintains acute myeloid leukemia through effects on isoform usage of epigenetic regulators including BRD4. Nat. Commun. 9:5378 (2018)
5.
Bowl, M.R.* et al.: A large scale hearing loss screen reveals an extensive unexplored genetic landscape for auditory dysfunction. Nat. Commun. 8:886 (2017)
6.
Dickinson, M.E.* et al.: Corrigendum: High-throughput discovery of novel developmental phenotypes. Nature 551, 398 (2017)
7.
Kaloff, C. et al.: Genome wide conditional mouse knockout resources. Drug Discov. Today, accepted (2017)
8.
Meehan, T.F.* et al.: Disease model discovery from 3,328 gene knockouts by The International Mouse Phenotyping Consortium. Nat. Genet. 49, 1231-1238 (2017)
9.
Bakker, B.* et al.: Single-cell sequencing reveals karyotype heterogeneity in murine and human malignancies. Genome Biol. 17:115 (2016)
10.
Dickinson, M.E.* et al.: High-throughput discovery of novel developmental phenotypes. Nature 537, 508-514 (2016)
11.
Maresch, R.* et al.: Multiplexed pancreatic genome engineering and cancer induction by transfection-based CRISPR/Cas9 delivery in mice. Nat. Commun. 7:10770 (2016)
12.
Weber, J.* et al.: CRISPR/Cas9 somatic multiplex-mutagenesis for high-throughput functional cancer genomics in mice. Proc. Natl. Acad. Sci. U.S.A. 112, 13982-13987 (2015)
13.
Schönhuber, N.* et al.: A next-generation dual-recombinase system for time- and host-specific targeting of pancreatic cancer. Nat. Med. 20, 1340-1347 (2014)
14.
Ayadi, A.* et al.: Mouse large-scale phenotyping initiatives: Overview of the European Mouse Disease Clinic (EUMODIC) and of the Wellcome Trust Sanger Institute mouse genetics project. Mamm. Genome 23, 600-610 (2012)
15.
Bradley, A.* et al.: The mammalian gene function resource: The International Knockout Mouse Consortium. Mamm. Genome 23, 580-586 (2012)
16.
Favor, J. et al.: Analysis of Pax6 contiguous gene deletions in the mouse, Mus musculus, identifies regions distinct from Pax6 responsible for extreme small-eye and belly-spotting phenotypes. Genetics 182, 1077-1088 (2009)