PuSH - Publication Server of Helmholtz Zentrum München

22 Records found.
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1.
Saleheen, D.* et al.: Loss of cardioprotective effects at the ADAMTS7 locus as a result of gene-smoking interactions. Circulation 135, 2336-2353 (2017)
2.
Golbus, J.R.* et al.: Common and rare genetic variation in CCR2, CCR5, or CX3CR1 and risk of atherosclerotic coronary heart disease and glucometabolic traits. Circ. Cardiovasc. Genet. 9, 250-258 (2016)
3.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N. Engl. J. Med. 374, 1134-1144 (2016)
4.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease (vol 374, pg 1134, 2016). N. Engl. J. Med. 374, 1898-1898 (2016)
5.
Interleukin 1 Genetics Consortium et al.: Cardiometabolic effects of genetic upregulation of the interleukin 1 receptor antagonist: A mendelian randomisation analysis. Lancet Diabet. Endocrinol. 3, 243-253 (2015)
6.
Ghosh, S.* et al.: Systems genetics analysis of genome-wide association study reveals novel associations between key biological processes and coronary artery disease. Arterioscler. Thromb. Vasc. Biol. 35, 1712-1722 (2015)
7.
Jansen, H.* et al.: Genetic variants primarily associated with type 2 diabetes are related to coronary artery disease risk. Atherosclerosis 241, 419-426 (2015)
8.
Nelson, C.P.* et al.: Genetically determined height and coronary artery disease. N. Engl. J. Med. 372, 1608-1618 (2015)
9.
Shungin, D.* et al.: New genetic loci link adipose and insulin biology to body fat distribution. Nature 518, 187-196 (2015)
10.
Dichgans, M.* et al.: Shared genetic susceptibility to ischemic stroke and coronary artery disease: A genome-wide analysis of common variants. Stroke 45, 24-36 (2014)
11.
Hartiala, J.* et al.: Comparative genome-wide association studies in mice and humans for trimethylamine N-oxide, a proatherogenic metabolite of choline and l-carnitine. Arterioscler. Thromb. Vasc. Biol. 34, 1307-1313 (2014)
12.
Erdmann, J.* et al.: Dysfunctional nitric oxide signalling increases risk of myocardial infarction. Nature 504, 432-436 (2013)
13.
Ho, J.E.* et al.: Common genetic variation at the IL1RL1 locus regulates IL-33/ST2 signaling. J. Clin. Invest. 123, 4208-4218 (2013)
14.
Lieb, W.* et al.: Genetic predisposition to higher blood pressure increases coronary artery disease risk. Hypertension 61, 995-1001 (2013)
15.
Davies, R.W.* et al.: A genome-wide association study for coronary artery disease identifies a novel susceptibility locus in the major histocompatibility complex. Circ. Cardiovasc. Genet. 5, 217-225 (2012)
16.
Schunkert, H.* et al.: Large-scale association analysis identifies 13 new susceptibility loci for coronary artery disease. Nat. Genet. 43, 333-340 (2011)
17.
Dupuis, J.* et al.: New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk. Nat. Genet. 42, 105-116 (2010)
18.
Liu, J.Z.* et al.: Meta-analysis and imputation refines the association of 15q25 with smoking quantity. Nat. Genet. 42, 436-440 (2010)
19.
Soranzo, N.* et al.: Common variants at 10 genomic loci influence hemoglobin A₁C levels via glycemic and nonglycemic pathways. Diabetes 59, 3229-3239 (2010)
20.
Teslovich, T.M.* et al.: Biological, clinical and population relevance of 95 loci for blood lipids. Nature 466, 707-713 (2010)