PuSH - Publication Server of Helmholtz Zentrum München

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1.
Saleheen, D.* et al.: Loss of cardioprotective effects at the ADAMTS7 locus as a result of gene-smoking interactions. Circulation 135, 2336-2353 (2017)
2.
Golbus, J.R.* et al.: Common and rare genetic variation in CCR2, CCR5, or CX3CR1 and risk of atherosclerotic coronary heart disease and glucometabolic traits. Circ. Cardiovasc. Genet. 9, 250-258 (2016)
3.
Keenan, T.* et al.: Causal assessment of serum urate levels in cardiometabolic diseases through a mendelian randomization study. J. Am. Coll. Cardiol. 67, 407-416 (2016)
4.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N. Engl. J. Med. 374, 1134-1144 (2016)
5.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease (vol 374, pg 1134, 2016). N. Engl. J. Med. 374, 1898-1898 (2016)
6.
Interleukin 1 Genetics Consortium et al.: Cardiometabolic effects of genetic upregulation of the interleukin 1 receptor antagonist: A mendelian randomisation analysis. Lancet Diabet. Endocrinol. 3, 243-253 (2015)
7.
Ghosh, S.* et al.: Systems genetics analysis of genome-wide association study reveals novel associations between key biological processes and coronary artery disease. Arterioscler. Thromb. Vasc. Biol. 35, 1712-1722 (2015)
8.
Jansen, H.* et al.: Genetic variants primarily associated with type 2 diabetes are related to coronary artery disease risk. Atherosclerosis 241, 419-426 (2015)
9.
Nelson, C.P.* et al.: Genetically determined height and coronary artery disease. N. Engl. J. Med. 372, 1608-1618 (2015)
10.
Dichgans, M.* et al.: Shared genetic susceptibility to ischemic stroke and coronary artery disease: A genome-wide analysis of common variants. Stroke 45, 24-36 (2014)
11.
Stitziel, N.O.* et al.: Inactivating mutations in NPC1L1 and protection from coronary heart disease. N. Engl. J. Med. 371, 2072-2082 (2014)
12.
Tragante, V.* et al.: Gene-centric meta-analysis in 87,736 individuals of European ancestry identifies multiple blood-pressure-related loci. Am. J. Hum. Genet. 94, 349-360 (2014)
13.
Global Lipids Genetics Consortium et al.: Discovery and refinement of loci associated with lipid levels. Nat. Genet. 45, 1274-1283 (2013)
14.
Codd, V.* et al.: Identification of seven loci affecting mean telomere length and their association with disease. Nat. Genet. 45, 422-427 (2013)
15.
Deloukas, P.* et al.: Large-scale association analysis identifies new risk loci for coronary artery disease. Nat. Genet. 45, 25-35 (2013)
16.
Do, R.* et al.: Common variants associated with plasma triglycerides and risk for coronary artery disease. Nat. Genet. 45, 1345-1352 (2013)
17.
Ganesh, S.K.* et al.: Loci influencing blood pressure identified using a cardiovascular gene-centric array. Hum. Mol. Genet. 22, 1663-1678 (2013)
18.
Ho, J.E.* et al.: Common genetic variation at the IL1RL1 locus regulates IL-33/ST2 signaling. J. Clin. Invest. 123, 4208-4218 (2013)
19.
Lieb, W.* et al.: Genetic predisposition to higher blood pressure increases coronary artery disease risk. Hypertension 61, 995-1001 (2013)
20.
Sabater-Lleal, M.* et al.: Multiethnic meta-analysis of genome-wide association studies in >100 000 subjects identifies 23 fibrinogen-associated loci but no strong evidence of a causal association between circulating fibrinogen and cardiovascular disease. Circulation 128, 1310-1324 (2013)