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1.
Kolder, I.C.* et al.: Analysis for genetic modifiers of disease severity in patients with long QT syndrome type 2. Circ. Cardiovasc. Genet. 8, 447-456 (2015)
2.
Arking, D.E.* et al.: Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization. Nat. Genet. 46, 826-836 (2014)
3.
Bezzina, C.R.* et al.: Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death. Nat. Genet. 45, 1044-1049 (2013)
4.
Kääb, S.* et al.: A large candidate gene survey identifies the KCNE1 D85N polymorphism as a possible modulator of drug-induced torsades de pointes. Circ. Cardiovasc. Genet. 5, 91-99 (2012)
5.
Kääb, S.* et al.: High density tagSNP candidate gene analysis identifies I-Ks as a major modulator of genetic susceptibility to drug induced long QT syndrome. Circulation 118, 2, S884-S884 (2008)
6.
Watanabe, H.* et al.: Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humans. J. Clin. Invest. 118, 2260-2268 (2008)