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1.
Moore, C.* et al.: Resequencing study confirms host defense and cell senescence gene variants contribute to the risk of idiopathic pulmonary fibrosis. Am. J. Respir. Crit. Care Med., accepted (2019)
2.
Sakornsakolpat, P.* et al.: Genetic landscape of chronic obstructive pulmonary disease identifies heterogeneous cell-type and phenotype associations. Nat. Genet. 51, 494-505 (2019)
3.
Shrine, N.* et al.: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries. Nat. Genet. 51, 481-493 (2019)
4.
Shrine, N.* et al.: Author Correction: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries (Nature Genetics, (2019), 51, 3, (481-493), 10.1038/s41588-018-0321-7). Nat. Genet., accepted (2019)
5.
Wain, L.V.* et al.: Genome-wide association analyses for lung function and chronic obstructive pulmonary disease identify new loci and potential druggable targets. Nat. Genet. 49, 416-425 (2017)
6.
Putman, R.K.* et al.: Association between interstitial lung abnormalities and all-cause mortality. JAMA 315, 672-681 (2016)
7.
Silverman, E.K.* et al.: Opportunities and challenges in the genetics of COPD 2010: An International COPD Genetics Conference report. COPD-J. Chronic Obstr. Pulm. Dis. 8, 121-135 (2011)
8.
Lasky-Su, J.* et al.: On the replication of genetic associations: Timing can be everything! Am. J. Hum. Genet. 82, 849-858 (2008)
9.
Lyon, H.N.* et al.: The association of a SNP upstream of INSIG2 with body mass index is reproduced in several but not all cohorts. PLoS Genet. 3, 0627-0633:e61 (2007)
10.
Raby, B.A.* et al.: Association of vitamin D receptor gene polymorphisms with childhood and adult asthma. Am. J. Respir. Crit. Care Med. 170, 1057-1065 (2004)