PuSH - Publication Server of Helmholtz Zentrum München

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1.
Sakornsakolpat, P.* et al.: Genetic landscape of chronic obstructive pulmonary disease identifies heterogeneous cell-type and phenotype associations. Nat. Genet. 51, 494-505 (2019)
2.
Shrine, N.* et al.: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries. Nat. Genet. 51, 481-493 (2019)
3.
Shrine, N.* et al.: Author Correction: New genetic signals for lung function highlight pathways and chronic obstructive pulmonary disease associations across multiple ancestries (Nature Genetics, (2019), 51, 3, (481-493), 10.1038/s41588-018-0321-7). Nat. Genet., accepted (2019)
4.
Jackson, V.E.* et al.: Meta-analysis of exome array data identifies six novel genetic loci for lung function. Wellcome Open Res. 3:4 (2018)
5.
Ji, X.* et al.: Identification of susceptibility pathways for the role of chromosome 15q25.1 in modifying lung cancer risk. Nat. Commun. 9:3221 (2018)
6.
Gref, A.* et al.: Genome-wide interaction analysis of air pollution exposure and childhood asthma with functional follow-up. Am. J. Respir. Crit. Care Med. 195, 1373-1383 (2017)
7.
McKay, J.D.* et al.: Large-scale association analysis identifies new lung cancer susceptibility loci and heterogeneity in genetic susceptibility across histological subtypes. Nat. Genet. 49, 1126-1132 (2017)
8.
Wain, L.V.* et al.: Genome-wide association analyses for lung function and chronic obstructive pulmonary disease identify new loci and potential druggable targets. Nat. Genet. 49, 416-425 (2017)
9.
Dvorkin-Gheva, A.* et al.: Total particulate matter concentration skews cigarette smoke’s gene expression profile. ERJ Open Res. 2:00029-2016 (2016)
10.
Fehringer, G.* et al.: Cross-cancer genome-wide analysis of lung, ovary, breast, prostate and colorectal cancer reveals novel pleiotropic associations. Cancer Res. 76, 5103-5114 (2016)
11.
Artigas, M.S.* et al.: Sixteen new lung function signals identified through 1000 Genomes Project reference panel imputation. Nat. Commun. 6:8658 (2015)
12.
Gharib, S.A.* et al.: Integrative pathway genomics of lung function and airflow obstruction. Hum. Mol. Genet. 24, 6836-6848 (2015)
13.
Obeidat, M.* et al.: Molecular mechanisms underlying variations in lung function: A systems genetics analysis. Lancet Resp. Med. 3, 782-795 (2015)
14.
Wain, L.V.* et al.: Novel insights into the genetics of smoking behaviour, lung function, and chronic obstructive pulmonary disease (UK BiLEVE): A genetic association study in UK Biobank. Lancet Resp. Med. 3, 769-781 (2015)
15.
Hancock, D.B.* et al.: Genome-wide joint meta-analysis of SNP and SNP-by-smoking interaction identifies novel loci for pulmonary function. PLoS Genet. 8:e1003098 (2013)
16.
Thun, G.A.* et al.: Causal and synthetic associations of variants in the SERPINA gene cluster with alpha1-antitrypsin serum levels. PLoS Genet. 9:e1003585 (2013)
17.
Artigas, M.S.* et al.: Genome-wide association and large-scale follow up identifies 16 new loci influencing lung function. Nat. Genet. 43, 1082-1090 (2011)
18.
Obeidat, M.* et al.: A comprehensive evaluation of potential lung function associated genes in the SpiroMeta general population sample. PLoS ONE 6:e19382 (2011)
19.
Soler Artigas, M.* et al.: Effect of five genetic variants associated with lung function on the risk of chronic obstructive lung disease, and their joint effects on lung function. Am. J. Respir. Crit. Care Med. 184, 786-795 (2011)
20.
Repapi, E.* et al.: Genome-wide association study identifies five loci associated with lung function. Nat. Genet. 42, 36-44 (2010)