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1.
Ma, J.* et al.: A peripheral blood DNA methylation signature of hepatic fat reveals a potential causal pathway for non-alcoholic fatty liver disease. Diabetes 68, 1073-1083 (2019)
2.
Ward-Caviness, C.K. et al.: Mendelian randomization evaluation of causal effects of fibrinogen on incident coronary heart disease. PLoS ONE 14:e0216222 (2019)
3.
Yao, C.* et al.: Genome-wide mapping of plasma protein QTLs identifies putatively causal genes and pathways for cardiovascular disease. Nat. Commun. 9:3268 (2018)
4.
Yao, C.* et al.: Genome-wide mapping of plasma protein QTLs identifies putatively causal genes and pathways for cardiovascular disease (vol 9, 3268, 2018). Nat. Commun. 9:3853 (2018)
5.
Shungin, D.* et al.: New genetic loci link adipose and insulin biology to body fat distribution. Nature 518, 187-196 (2015)
6.
Global Lipids Genetics Consortium et al.: Discovery and refinement of loci associated with lipid levels. Nat. Genet. 45, 1274-1283 (2013)
7.
Deloukas, P.* et al.: Large-scale association analysis identifies new risk loci for coronary artery disease. Nat. Genet. 45, 25-35 (2013)
8.
den Hoed, M.* et al.: Identification of heart rate-associated loci and their effects on cardiac conduction and rhythm disorders. Nat. Genet. 45, 621-631 (2013)
9.
Dimas, A.S.* et al.: Impact of type 2 diabetes susceptibility variants on quantitative glycemic traits reveals mechanistic heterogeneity. Diabetes 63, 2158-2171 (2013)
10.
Do, R.* et al.: Common variants associated with plasma triglycerides and risk for coronary artery disease. Nat. Genet. 45, 1345-1352 (2013)
11.
Song, C.* et al.: Genetic variants from lipid-related pathways and risk for incident myocardial infarction. PLoS ONE 8:e60454 (2013)
12.
Kilpeläinen, T.O.* et al.: Genetic variation near IRS1 associates with reduced adiposity and an impaired metabolic profile. Nat. Genet. 43, 753-760 (2011)