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Kahle, M. et al.: High fat diet-induced modifications in membrane lipid and mitochondrial-membrane protein signatures precede the development of hepatic insulin resistance in mice. Mol. Metab. 4, 39-50 (2015)
Ly-Verdú, S. et al.: Combining metabolomic non-targeted GC×GC-ToF-MS analysis and chemometric ASCA-based study of variances to assess dietary influence on type 2 diabetes development in a mouse model. Anal. Bioanal. Chem. 407, 343-354 (2015)
Schäfer, A. et al.: The epoxyeicosatrienoic acid pathway enhances hepatic insulin signaling and is repressed in insulin-resistant mouse liver. Mol. Cell. Proteomics 14, 2764-2774 (2015)
Ly-Verdú, S. et al.: The impact of blood on liver metabolite profiling - a combined metabolomic and proteomic approach. Biomed. Chromatogr. 28, 231-240 (2014)
Walker, A. et al.: Distinct signatures of host-microbial meta-metabolome and gut microbiome in two C57BL/6 strains under high-fat diet. ISME J. 8, 2380-2396 (2014)
Kahle, M. et al.: Phenotypic comparison of common mouse strains developing high-fat diet-induced hepatosteatosis. Mol. Metab. 2, 435-446 (2013)
Venit, T.* et al.: Mouse nuclear myosin 1 knock-out shows interchangeability and redundancy of myosin isoforms in the cell nucleus. PLoS ONE 8:e61406 (2013)
von Toerne, C. et al.: Apoe, Mbl2 and Psp plasma protein levels correlate with diabetic phenotype in NZO mice - an optimized rapid workflow for SRM-based quantification. J. Proteome Res. 12, 1331-1343 (2013)
Fuchs, H. et al.: Mouse genetics and metabolic mouse phenotyping. In: Suhre, K.* [Eds.]: Genetics Meets Metabolomics: from Experiment to Systems Biology. Springer, 2012. 85-106
Fuchs, H. et al.: Innovations in phenotyping of mouse models in the German Mouse Clinic. Mamm. Genome 23, 611-622 (2012)
Schäfer, A. et al.: Two-dimensional peptide separation improving sensitivity of selected reaction monitoring-based quantitative proteomics in mouse liver tissue: Comparing off-gel electrophoresis and strong cation exchange chromatography. Anal. Chem. 84, 8853-8862 (2012)
Kiefer, F.W.* et al.: Osteopontin deficiency protects against obesity-induced hepatic steatosis and attenuates glucose production in mice. Diabetologia 54, 2132-2142 (2011)