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Adriaens, M.E.* et al.: Systems genetics approaches in rat identify novel genes and gene networks associated with cardiac conduction. J. Am. Heart Assoc. 7:e009243 (2018)
Ashar, F.N.* et al.: A comprehensive evaluation of the genetic architecture of sudden cardiac arrest. Eur. Heart J. 39, 3961-3969 (2018)
Heinig, M. et al.: Natural genetic variation of the cardiac transcriptome in non-diseased donors and patients with dilated cardiomyopathy. Genome Biol. 18:170 (2017)
Milano, A.* et al.: Sudden cardiac arrest and rare genetic variants in the community. Circ. Cardiovasc. Genet. 9, 147-153 (2016)
van der Harst, P.* et al.: 52 Genetic loci influencing myocardial mass. J. Am. Coll. Cardiol. 68, 1435-1448 (2016)
Wilde, A.A.* et al.: Clinical aspects of type 3 long QT syndrome: An international multicenter study. Circulation 134, 872-882 (2016)
Behr, E.R.* et al.: Role of common and rare variants in SCN10A: Results from the Brugada syndrome QRS locus gene discovery collaborative study. Cardiovasc. Res. 106, 520-529 (2015)
Kolder, I.C.* et al.: Analysis for genetic modifiers of disease severity in patients with long QT syndrome type 2. Circ. Cardiovasc. Genet. 8, 447-456 (2015)
Arking, D.E.* et al.: Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization. Nat. Genet. 46, 826-836 (2014)
Tragante, V.* et al.: Gene-centric meta-analysis in 87,736 individuals of European ancestry identifies multiple blood-pressure-related loci. Am. J. Hum. Genet. 94, 349-360 (2014)
Bezzina, C.R.* et al.: Common variants at SCN5A-SCN10A and HEY2 are associated with Brugada syndrome, a rare disease with high risk of sudden cardiac death. Nat. Genet. 45, 1044-1049 (2013)
Amin, A.S.* et al.: Variants in the 3' untranslated region of the KCNQ1-encoded Kv7.1 potassium channel modify disease severity in patients with type 1 long QT syndrome in an allele-specific manner. Eur. Heart J. 33, 714-723 (2012)
Asselbergs, F.W.* et al.: Large-scale gene-centric meta-analysis across 32 studies identifies multiple lipid loci. Am. J. Hum. Genet. 91, 823-838 (2012)
Kääb, S.* et al.: A large candidate gene survey identifies the KCNE1 D85N polymorphism as a possible modulator of drug-induced torsades de pointes. Circ. Cardiovasc. Genet. 5, 91-99 (2012)
Bezzina, C.R.* et al.: Genome-wide association study identifies a susceptibility locus at 21q21 for ventricular fibrillation in acute myocardial infarction. Nat. Genet. 42, 688-691 (2010)
Horn, D.* et al.: Identification of FOXP1 deletions in three unrelated patients with mental retardation and significant speech and language deficits. Hum. Mutat. 31, E1851-E1860 (2010)
Kääb, S.* et al.: High density tagSNP candidate gene analysis identifies I-Ks as a major modulator of genetic susceptibility to drug induced long QT syndrome. Circulation 118, 2, S884-S884 (2008)
Watanabe, H.* et al.: Sodium channel β1 subunit mutations associated with Brugada syndrome and cardiac conduction disease in humans. J. Clin. Invest. 118, 2260-2268 (2008)
Yang, P.* et al.: Polymorphisms in the cardiac sodium channel promoter displaying variant in vitro expression activity. Eur. J. Hum. Genet. 16, 350-357 (2008)