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1.
Styrkarsdottir, U.* et al.: GWAS of bone size yields twelve loci that also affect height, BMD, osteoarthritis or fractures. Nat. Commun. 10:2054 (2019)
2.
Styrkarsdottir, U.* et al.: Erratum: Publisher Correction: GWAS of bone size yields twelve loci that also affect height, BMD, osteoarthritis or fractures (Nature communications (2019) 10 1 (2054)). Nat. Commun. 10:2358 (2019)
3.
Scott, R.A.* et al.: An expanded genome-wide association study of type 2 diabetes in Europeans. Diabetes 66, 2888-2902 (2017)
4.
Gaulton, K.J.* et al.: Genetic fine mapping and genomic annotation defines causal mechanisms at type 2 diabetes susceptibility loci. Nat. Genet. 47, 1415-1425 (2015)
5.
Flannick, J.* et al.: Loss-of-function mutations in SLC30A8 protect against type 2 diabetes. Nat. Genet. 46, 357-363 (2014)
6.
Morris, A.P.* et al.: Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nat. Genet. 44, 981-990 (2012)
7.
Perry, J.R.* et al.: Stratifying type 2 diabetes cases by BMI identifies genetic risk variants in LAMA1 and enrichment for risk variants in lean compared to obese cases. PLoS Genet. 8:e1002741 (2012)
8.
Dupuis, J.* et al.: New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk. Nat. Genet. 42, 105-116 (2010)
9.
Voight, B.F.* et al.: Twelve type 2 diabetes susceptibility loci identified through large-scale association analysis. Nat. Genet. 42, 579-589 (2010)
10.
Kong, A.* et al.: Parental origin of sequence variants associated with complex diseases. Nature 462, 868-874 (2009)