PuSH - Publication Server of Helmholtz Zentrum München

11 Records found.
Zum Exportieren der Ergebnisse bitte einloggen.
Lay all publications on this page into basket
1.
Scott, R.A.* et al.: An expanded genome-wide association study of type 2 diabetes in Europeans. Diabetes 66, 2888-2902 (2017)
2.
Winkler, T.W.* et al.: Correction: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 12:e1006166 (2016)
3.
Gaulton, K.J.* et al.: Genetic fine mapping and genomic annotation defines causal mechanisms at type 2 diabetes susceptibility loci. Nat. Genet. 47, 1415-1425 (2015)
4.
Winkler, T.W.* et al.: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 11:e1005378 (2015)
5.
Jarick, I.* et al.: Genome-wide analysis of rare copy number variations reveals PARK2 as a candidate gene for attention-deficit/hyperactivity disorder. Mol. Psychiatry 19, 115–121 (2014)
6.
Morris, A.P.* et al.: Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nat. Genet. 44, 981-990 (2012)
7.
Hinney, A.* et al.: Genome-wide association study in German patients with attention deficit/hyperactivity disorder. Am. J. Med. Genet. B 156B, 888-897 (2011)
8.
Karakas, M.* et al.: Copeptin does not add diagnostic information to high-sensitivity troponin T in low- to intermediate-risk patients with acute chest pain: Results from the Rule Out Myocardial Infarction by Computed Tomography (ROMICAT) study. Clin. Chem. 57, 1137-1145 (2011)
9.
Meyer, J. et al.: Dietary patterns, subclinical inflammation, incident coronary heart disease and mortality in middle-aged men from the MONICA/KORA Augsburg cohort study. Eur. J. Clin. Nutr. 65, 800-807 (2011)
10.
Stocking, C.* et al.: Cell and virus genetics at the roots of gene therapy, retrovirology, and hematopoietic stem cell biology: Wolfram Ostertag (1937-2010). Hum. Gene Ther. 21, 1501-1503 (2010)
11.
Meyer, J.* et al.: A missense mutation in a novel gene encoding a putative cation channel is associated with catatonic schizophrenia in a large pedigree. Mol. Psychiatry 6, 302-306 (2001)