PuSH - Publication Server of Helmholtz Zentrum München

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1.
Bentley, A.R.* et al.: Multi-ancestry genome-wide gene–smoking interaction study of 387,272 individuals identifies new loci associated with serum lipids. Nat. Genet. 51, 636-648 (2019)
2.
de Vries, P.S.* et al.: Multi-ancestry Genome-Wide Association study of lipid levels incorporating gene-alcohol interactions. Am. J. Epidemiol. 188, 1033-1054 (2019)
3.
Flannick, J.* et al.: Exome sequencing of 20,791 cases of type 2 diabetes and 24,440 controls. Nature 570, 71-76 (2019)
4.
Justice, A.E.* et al.: Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution. Nat. Genet. 51, 452–469 (2019)
5.
Sung, Y.J.* et al.: A multi-ancestry genome-wide study incorporating gene-smoking interactions identifies multiple new loci for pulse pressure and mean arterial pressure. Hum. Mol. Genet., accepted (2019)
6.
Flannick, J.* et al.: Sequence data and association statistics from 12,940 type 2 diabetes cases and controls. Sci. Data 5:180002 (2018)
7.
Mahajan, A.* et al.: Fine-mapping type 2 diabetes loci to single-variant resolution using high-density imputation and islet-specific epigenome maps. Nat. Genet. 50, 1505-1513 (2018)
8.
Sung, Y.J.* et al.: A large-scale multi-ancestry genome-wide study accounting for smoking behavior identifies multiple significant loci for blood pressure. Am. J. Hum. Genet. 102, 375-400 (2018)
9.
Turcot, V.* et al.: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity. Nat. Genet. 50, 26-41 (2018)
10.
Turcot, V.* et al.: Protein-altering variants associated with body mass index implicate pathways that control energy intake and expenditure in obesity (vol 50, pg 765, 2017). Nat. Genet. 50, 764-768 (2018)
11.
Flannick, J.* et al.: Sequence data and association statistics from 12,940 type 2 diabetes cases and controls. Sci. Data 4:170179 (2017)
12.
Manning, A.* et al.: A low-frequency inactivating Akt2 variant enriched in the Finnish population is associated with fasting insulin levels and type 2 diabetes risk. Diabetes 66, 2019-2032 (2017)
13.
Marouli, E.* et al.: Rare and low-frequency coding variants alter human adult height. Nature 542, 186-190 (2017)
14.
Saleheen, D.* et al.: Loss of cardioprotective effects at the ADAMTS7 locus as a result of gene-smoking interactions. Circulation 135, 2336-2353 (2017)
15.
Wood, A.R.* et al.: A genome-wide association study of IVGTT-based measures of first-phase insulin secretion refines the underlying physiology of type 2 diabetes variants. Diabetes 66, 2296-2309 (2017)
16.
Fuchsberger, C.* et al.: The genetic architecture of type 2 diabetes. Nature 536, 41-47 (2016)
17.
Flannick, J.* et al.: Loss-of-function mutations in SLC30A8 protect against type 2 diabetes. Nat. Genet. 46, 357-363 (2014)
18.
Ng, M.C.Y.* et al.: Meta-analysis of genome-wide association studies in African Americans provides insights into the genetic architecture of type 2 diabetes. PLoS Genet. 10:e100451 (2014)
19.
Saxena, R.* et al.: Large-scale gene-centric meta-analysis across 39 studies identifies type 2 diabetes loci. Am. J. Hum. Genet. 90, 410-425 (2012)
20.
Zaitlen, N.* et al.: Informed conditioning on clinical covariates increases power in case-control association studies. PLoS Genet. 8:e1003032 (2012)