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1.
Mroz, P.A.* et al.: Optimized GIP analogs promote body weight lowering in mice through GIPR agonism not antagonism. Mol. Metab. 20, 51-62 (2019)
2.
Kim, T.* et al.: Glucagon receptor signaling regulates energy metabolism via hepatic farnesoid X receptor and fibroblast growth factor 21. Diabetes 67, 1773-1782 (2018)
3.
Ryan, K.K.* et al.: Dietary manipulations that induce ketosis activate the HPA axis in male rats and mice: A potential role for fibroblast growth factor-21. Endocrinology 159, 400-413 (2018)
4.
Knerr, P.J.* et al.: Optimization of peptide-based polyagonists for treatment of diabetes and obesity. Bioorg. Med. Chem. 26, 2873-2881 (2017)
5.
Quarta, C. et al.: Molecular integration of incretin and glucocorticoid action reverses immunometabolic dysfunction and obesity. Cell Metab. 26, 620-632.e6 (2017)
6.
Finan, B. ; Müller, T.D. ; Clemmensen, C. ; Perez-Tilve, D.* & Tschöp, M.H.: Reappraisal of GIP pharmacology for metabolic diseases. Trends Mol. Med. 22, 359-376 (2016)
7.
Finan, B. et al.: Chemical hybridization of glucagon and thyroid hormone optimizes therapeutic Impact for metabolic disease. Cell 167, 843-857.e14 (2016)
8.
Kabra, D.G. et al.: Hypothalamic leptin action is mediated by histone deacetylase 5. Nat. Commun. 7:10782 (2016)
9.
Raab, S.* et al.: Incretin-like effects of small molecule trace amine-associated receptor 1 agonists. Mol. Metab. 5, 47-56 (2016)
10.
Sánchez-Garrido, M.A. et al.: Fibroblast activation protein (FAP) as a novel metabolic target. Mol. Metab. 5, 1015-24 (2016)
11.
Tschöp, M.H. et al.: Unimolecular polypharmacy for treatment of diabetes and obesity. Cell Metab. 24, 51-62 (2016)
12.
Finan, B. et al.: A rationally designed monomeric peptide triagonist corrects obesity and diabetes in rodents. J. Nat. Med. 21, 27-36 (2015)
13.
Müller, T.D. et al.: Ghrelin. Mol. Metab. 4, 437–460 (2015)
14.
Pfluger, P.T. et al.: Calcineurin links mitochondrial elongation with energy metabolism. Cell Metab. 22, 838-850 (2015)
15.
Gao, Y. et al.: Hormones and diet, but not body weight, control hypothalamic microglial activity. Glia 62, 17-25 (2014)
16.
Habegger, K.M.* et al.: Duodenal nutrient exclusion improves metabolic syndrome and stimulates villus hyperplasia. Gut 63, 1238-1246 (2014)
17.
Habegger, K.M.* et al.: GLP-1R responsiveness predicts individual gastric bypass efficacy on glucose tolerance in rats. Diabetes 63, 505-513 (2014)
18.
Heppner, K.M.* et al.: Both acyl and des-acyl ghrelin regulate adiposity and glucose metabolism via CNS ghrelin receptors. Diabetes 63, 122-131 (2014)
19.
Al-Massadi, O.* ; Heppner, K.M.* ; Nogueiras, R.A.* ; Perez-Tilve, D.* & Tschöp, M.H.: Ghrelin. In: Kastin, A.* [Eds.]: Handbook of Biologically Active Peptides. Amsterdam: Elsevier, 2013. 1104-1110
20.
Finan, B. et al.: Unimolecular dual incretins maximize metabolic benefits in rodents, monkeys, and humans. Sci. Transl. Med. 5:209ra151 (2013)