PuSH - Publication Server of Helmholtz Zentrum München

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1.
Mühlig, Y.* et al.: Psychosoziale Charakterisierung und Integrative Versorgung arbeitsloser Jugendlicher mit extremer Adipositas – ein Modellprojekt. Psychother. Psychosom. Med. Psychol., accepted (2019)
2.
Bühlmeier, J.* et al.: Dietary acid load and mental health outcomes in children and adolescents: Results from the GINIplus and LISA birth cohort studies. Nutrients 10:582 (2018)
3.
Giuranna, J.* et al.: The effect of SH2B1 variants on expression of leptin- and insulin-induced pathways in murine hypothalamus. Obes. Facts 11, 93-108 (2018)
4.
Huckins, L.M.* et al.: Investigation of common, low-frequency and rare genome-wide variation in anorexia nervosa. Mol. Psychiatry 23, 1169-1180 (2018)
5.
Huckins, L.M.* et al.: Correction: Investigation of common, low-frequency and rare genome-wide variation in anorexia nervosa. Mol. Psychiatry 23:1 (2018)
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Kesselmeier, M.* et al.: High-throughput DNA methylation analysis in anorexia nervosa confirms TNXB hypermethylation. World J. Biol. Psychiatry 19, 187-199 (2018)
7.
Lennerz, B.S.* et al.: Do adolescents with extreme obesity differ according to previous treatment seeking behavior? The Youth with Extreme obesity Study (YES) cohort. Int. J. Obes., accepted (2018)
8.
Hinney, A.* et al.: Evidence for three genetic loci involved in both anorexia nervosa risk and variation of body mass index. Mol. Psychiatry 22, 192-201 (2017)
9.
Hinney, A.* et al.: Evidence for three genetic loci involved in both anorexia nervosa risk and variation of body mass index (vol 22, pg 192, 2017). Mol. Psychiatry 22, 321-322 (2017)
10.
Lennerz, B.* et al.: Medical and psychosocial implications of adoelscent extreme obesity- acceptance and effects of structured care program - Yes Study. A consortium of the BMBF. Horm. Res. Paediatr. 88, 161-161 (2017)
11.
Li, D.* et al.: A genome-wide association study of anorexia nervosa suggests a risk locus implicated in dysregulated leptin signaling. Sci. Rep. 7:3847 (2017)
12.
Mühlig, Y.* et al.: A structured, manual-based low-level intervention vs. treatment as usual evaluated in a randomized controlled trial for adolescents with extreme obesity - the STEREO trial. Obes. Facts 10, 341-352 (2017)
13.
Mooney, M.A.* et al.: Pathway analysis in attention deficit hyperactivity disorder: An ensemble approach. Am. J. Med. Genet. B 171, 815-826 (2016)
14.
Volckmar, A.L.* et al.: Analysis of genes involved in body weight regulation by targeted re-sequencing. PLoS ONE 11:e0147904 (2016)
15.
Wang, H.J.* et al.: Association of common variants identified by recent genome-wide association studies with obesity in Chinese children: A case-control study. BMC Med. Genet. 17:7 (2016)
16.
Nead, K.T.* et al.: Contribution of common non-synonymous variants in PCSK1 to body mass index variation and risk of obesity: A systematic review and meta-analysis with evidence from up to 331 175 individuals. Hum. Mol. Genet. 24, 3582-3594 (2015)
17.
Volckmar, A.L.* et al.: Fine mapping of a GWAS-derived obesity candidate region on chromosome 16p11.2. PLoS ONE 10:e0125660 (2015)
18.
Boraska, V.* et al.: A genome-wide association study of anorexia nervosa. Mol. Psychiatry 19, 1085-1094 (2014)
19.
Hinney, A.* et al.: Genetic variation at the CELF1 (CUGBP, elav-like family member 1 gene) locus is genome-wide associated with Alzheimer's disease and obesity. Am. J. Med. Genet. B 165, 283-293 (2014)
20.
Hoggart, C.J.* et al.: Novel approach identifies SNPs in SLC2A10 and KCNK9 with evidence for parent-of-origin effect on Body Mass Index. PLoS Genet. 10, 1-12:e1004508 (2014)