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1.
Gerst, F. et al.: Fetuin-A impairs islet differentiation and function via inhibition of TGFbeta-1 signalling. Diabetologia 61, S205-S206 (2018)
2.
Hinrichs, A.* et al.: Growth hormone receptor-deficient pigs resemble the pathophysiology of human Laron syndrome and reveal altered activation of signaling cascades in the liver. Mol. Metab. 11, 113-128 (2018)
3.
Blutke, A.* et al.: The Munich MIDY Pig Biobank - A unique resource for studying organ crosstalk in diabetes. Mol. Metab. 6, 931-940 (2017)
4.
Kemter, E.* et al.: INS-eGFP transgenic pigs: A novel reporter system for studying maturation, growth and vascularisation of neonatal islet-like cell clusters. Diabetologia 60, 1152-1156 (2017)
5.
Kumar, S.* et al.: Standardized, systemic phenotypic analysis reveals kidney dysfunction as main alteration of Kctd1I27N mutant mice. J. Biomed. Sci. 24:57 (2017)
6.
Kourtzelis, I.* et al.: Developmental endothelial locus-1 modulates platelet-monocyte interactions and instant blood-mediated inflammatory reaction in islet transplantation. Thromb. Haemost. 115, 781-788 (2016)
7.
Kumar, S.* et al.: Generation and standardized, systemic phenotypic analysis of Pou3f3L423P mutant mice. PLoS ONE 11:e0150472 (2016)
8.
Ivanova-Cederstroem, A. et al.: Generation of the transgenic INS-SNAP (SOFIA) pig for the high-throughput high-content study of insulin granule biogenesis and turnover in isolated islets. Diabetologia 58, S217 (2015)
9.
Kemter, E.* et al.: INS-eGFP transgenic piglets provide a novel tool for studying maturation and vascularization of Neonatal Islet Cell Clusters (NICCs) in vivo. Xenotransplantation 22, S62-S63 (2015)
10.
Kemter, E.* et al.: INS-EGFP transgenic piglets provide a novel tool for studying maturation and vascularization of Neonatal Islet Cell Clusters (NICCs) in vivo. Transplantation 99, S99 (2015)
11.
Le, M.* et al.: Detailed morphological analyses of a novel mouse model of site-specific glomerular thrombotic microangiopathy. Nephrol. Dial. Transplant. 30:SP045 (2015)
12.
Horsch, M. et al.: Uromodulin retention in thick ascending limb of Henle's loop affects SCD1 in neighboring proximal tubule: Renal transcriptome studies in mouse models of uromodulin-associated kidney disease. PLoS ONE 9:e113125 (2014)
13.
Kemter, E.* et al.: No amelioration of uromodulin maturation and trafficking defect by sodium-4-phenylbutyrate in vivo: Studies in mouse models of uromodulin-associated kidney disease. J. Biol. Chem. 289, 10715-10726 (2014)
14.
Kemter, E.* et al.: Standardized, systemic phenotypic analysis of Slc12a1 I299F mutant mice. J. Biomed. Sci. 21:68 (2014)
15.
Kemter, E.* et al.: Type of uromodulin mutation and allelic status influence onset and severity of uromodulin-associated kidney disease in mice. Hum. Mol. Genet. 22, 4148-4163 (2013)
16.
Kemter, E.* et al.: Standardized, systemic phenotypic analysis of UmodC93F and UmodA227T mutant mice. PLoS ONE 8:e78337 (2013)
17.
Fuchs, H. et al.: Mouse phenotyping. Methods 53, 120-135 (2011)
18.
Kemter, E.* et al.: Mutation of the Na⁺-K⁺-2Cl‾ cotransporter NKCC2 in mice is associated with severe polyuria and a urea-selective concentrating defect without hyperreninemia. Am. J. Physiol.-Renal Physiol. 298, F1405-F1415 (2010)
19.
van Bürck, L.* et al.: Phenotypic and pathomorphological characteristics of a novel mutant mouse model for maturity-onset diabetes of the young type 2 (MODY 2). Am. J. Physiol. Endocrinol. Metab. 298, E512-E523 (2010)
20.
Kemter, E.* et al.: Novel missense mutation of uromodulin in mice causes renal dysfunction with alterations in urea handling, energy, and bone metabolism. Am. J. Physiol.-Renal Physiol. 297, F1391-F1398 (2009)