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1.
Hess, J. et al.: A five-microRNA signature predicts survival and disease control of patients with head and neck cancer negative for HPV-infection. Clin. Cancer Res. 25, 1505-1516 (2019)
2.
Hess, J. et al.: A five-microRNA-signature predicts recurrence and survival in HPV-negative HNSCC. Strahlenther. Onkol. 195, S25-S25 (2019)
3.
Liu, X.* et al.: Epithelial-type systemic breast carcinoma cells with a restricted mesenchymal transition are a major source of metastasis. Sci. Adv. 5:eaav4275 (2019)
4.
Caldwell, R.B. ; Braselmann, H. ; Heuer, S. ; Schötz, U.* & Zitzelsberger, H.: Gain-of-function analysis of cis-acting diversification elements in DT40 cells. Immunol. Cell Biol. 96, 948-957 (2018)
5.
Dalke, C. et al.: Lifetime study in mice after acute low-dose ionizing radiation: A multifactorial study with special focus on cataract risk. Radiat. Environ. Biophys. 57, 99-113 (2018)
6.
Wilke, C. et al.: Expression of miRNA-26b-5p and its target TRPS1 is associated with radiation exposure in post-Chernobyl breast cancer. Int. J. Cancer 142, 573-583 (2018)
7.
Wilke, C. et al.: A genomic copy number signature predicts radiation exposure in post-Chernobyl breast cancer. Int. J. Cancer 143, 1505-1515 (2018)
8.
Braselmann, H.: Heterogeneous correlation of multi-level omics data for the consideration of inter-tumoural heterogeneity. In: (Extended Abstracts Fall 2015). 2017. 71-75 (Trends Math. ; 7)
9.
Hess, J. et al.: Genomic amplification of Fanconi anemia complementation group A (FancA) in head and neck squamous cell carcinoma (HNSCC): Cellular mechanisms of radioresistance and clinical relevance. Cancer Lett. 386, 87-99 (2017)
10.
Caldwell, R.B. et al.: Positive Cofactor 4 (PC4) is critical for DNA repair pathway re-routing in DT40 cells. Sci. Rep. 6:28890 (2016)
11.
Huber, M. et al.: Cyr61 and YB-1 are novel interacting partners of uPAR and elevate the malignancy of triple-negative breast cancer. Oncotarget 7, 44062-44075 (2016)
12.
Huber, M. et al.: uPAR enhances malignant potential of triple-negative breast cancer by directly interacting with uPA and IGF1R. BMC Cancer 16:615 (2016)
13.
Michna, A. et al.: Natural cubic spline regression modeling followed by dynamic network reconstruction for the identification of radiation-sensitivity gene association networks from time-course transcriptome data. PLoS ONE 11:e0160791 (2016)
14.
Penterling, C.* et al.: Depletion of histone demethylase Jarid1A resulting in histone hyperacetylation and radiation sensitivity does not affect DNA double-strand break repair. PLoS ONE 11:e0156599 (2016)
15.
Abend, M.* et al.: Association of radiation-induced genes with noncancer chronic diseases in Mayak workers occupationally exposed to prolonged radiation. Radiat. Res. 183, 249-261 (2015)
16.
Aubele, M. et al.: uPA receptor and its interaction partners: Impact as potential therapeutic targets in triple-negative breast cancer. J. Clin. Oncol. 33:150 (2015)
17.
Braselmann, H. ; Michna, A. ; Hess, J. & Unger, K.: CFAssay: Statistical analysis of the colony formation assay. Radiat. Oncol. 10:223 (2015)
18.
Gimenez-Aznar, I. et al.: Fanconi anemia, complementation group A (FancA) overexpression confers radioresistance to oral keratinocytes. Eur. J. Cancer 51, S121 (2015)
19.
Hess, J. et al.: Signalling networks associated with FancA mediated radioresistance in cells of head and neck squamous cell carcinoma. Strahlenther. Onkol. 191, S77 (2015)
20.
Huber, M. et al.: The impact of the uPAR system and its interaction partners as potential therapeutic targets in TNBC. Ann. Oncol. 26:67P (2015)