PuSH - Publication Server of Helmholtz Zentrum München

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1.
Justice, A.E.* et al.: Protein-coding variants implicate novel genes related to lipid homeostasis contributing to body-fat distribution. Nat. Genet. 51, 452–469 (2019)
2.
Lane, J.M.* et al.: Biological and clinical insights from genetics of insomnia symptoms. Nat. Genet. 51, 387-393 (2019)
3.
Mahajan, A.* et al.: Refining the accuracy of validated target identification through coding variant fine-mapping in type 2 diabetes. Nat. Genet. 50, 559-571 (2018)
4.
Braenne, I.* et al.: Genomic correlates of glatiramer acetate adverse cardiovascular effects lead to a novel locus mediating coronary risk. PLoS ONE 12:e0182999 (2017)
5.
Braenne, I.* et al.: A genomic exploration identifies mechanisms that may explain adverse cardiovascular effects of COX-2 inhibitors. Sci. Rep. 7:10252 (2017)
6.
Ferreira, M.A.* et al.: Shared genetic origin of asthma, hay fever and eczema elucidates allergic disease biology. Nat. Genet. 49, 1752-1757 (2017)
7.
Lu, X.* et al.: Exome chip meta-analysis identifies novel loci and East Asian-specific coding variants that contribute to lipid levels and coronary artery disease. Nat. Genet. 49, 1722–1730 (2017)
8.
Ehret, G.B.* et al.: The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals. Nat. Genet. 48, 1171-1184 (2016)
9.
Lu, Y.* et al.: New loci for body fat percentage reveal link between adiposity and cardiometabolic disease risk. Nat. Commun. 7:10495 (2016)
10.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N. Engl. J. Med. 374, 1134-1144 (2016)
11.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease (vol 374, pg 1134, 2016). N. Engl. J. Med. 374, 1898-1898 (2016)
12.
Surendran, P.* et al.: Trans-ancestry meta-analyses identify rare and common variants associated with blood pressure and hypertension. Nat. Genet. 48, 1151-1161 (2016)
13.
Zanoni, P.* et al.: Rare variant in scavenger receptor BI raises HDL cholesterol and increases risk of coronary heart disease. Science 351, 1166-1671 (2016)
14.
Locke, A.E.* et al.: Genetic studies of body mass index yield new insights for obesity biology. Nature 518, 197-206 (2015)
15.
Nelson, C.P.* et al.: Genetically determined height and coronary artery disease. N. Engl. J. Med. 372, 1608-1618 (2015)
16.
Shungin, D.* et al.: New genetic loci link adipose and insulin biology to body fat distribution. Nature 518, 187-196 (2015)
17.
Ganesh, S.K.* et al.: Effects of long-term averaging of quantitative blood pressure traits on the detection of genetic associations. Am. J. Hum. Genet. 95, 49-65 (2014)
18.
Lange, L.A.* et al.: Whole-exome sequencing identifies rare and low-frequency coding variants associated with LDL cholesterol. Am. J. Hum. Genet. 94, 233-245 (2014)
19.
Wood, A.R.* et al.: Defining the role of common variation in the genomic and biological architecture of adult human height. Nat. Genet. 46, 1173-1186 (2014)
20.
Global Lipids Genetics Consortium et al.: Discovery and refinement of loci associated with lipid levels. Nat. Genet. 45, 1274-1283 (2013)