PuSH - Publication Server of Helmholtz Zentrum München

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1.
Chen, G.B.* et al.: Across-cohort QC analyses of GWAS summary statistics from complex traits. Eur. J. Hum. Genet. 25, 137-146 (2017)
2.
Scott, R.A.* et al.: An expanded genome-wide association study of type 2 diabetes in Europeans. Diabetes 66, 2888-2902 (2017)
3.
Wain, L.V.* et al.: Genome-wide association analyses for lung function and chronic obstructive pulmonary disease identify new loci and potential druggable targets. Nat. Genet. 49, 416-425 (2017)
4.
Ehret, G.B.* et al.: The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals. Nat. Genet. 48, 1171-1184 (2016)
5.
Fuchsberger, C.* et al.: The genetic architecture of type 2 diabetes. Nature 536, 41-47 (2016)
6.
Kilpeläinen, T.O.* et al.: Genome-wide meta-analysis uncovers novel loci influencing circulating leptin levels. Nat. Commun. 7:10494 (2016)
7.
Lessard, S.* et al.: Testing the role of predicted gene knockouts in human anthropometric trait variation. Hum. Mol. Genet. 25, 2082-2092 (2016)
8.
Lu, Y.* et al.: New loci for body fat percentage reveal link between adiposity and cardiometabolic disease risk. Nat. Commun. 7:10495 (2016)
9.
Pattaro, C.* et al.: Genetic associations at 53 loci highlight cell types and biological pathways relevant for kidney function. Nat. Commun. 7:10023 (2016)
10.
Teumer, A.* et al.: Genome-wide association studies identify genetic loci associated with albuminuria in diabetes. Diabetes 65, 803-817 (2016)
11.
Coffee and Caffeine Genetics Consortium et al.: Genome-wide meta-analysis identifies six novel loci associated with habitual coffee consumption. Mol. Psychiatry 20, 647-656 (2015)
12.
Gaulton, K.J.* et al.: Genetic fine mapping and genomic annotation defines causal mechanisms at type 2 diabetes susceptibility loci. Nat. Genet. 47, 1415-1425 (2015)
13.
Joshi, P.K.* et al.: Directional dominance on stature and cognition in diverse human populations. Nature 523, 459-462 (2015)
14.
Locke, A.E.* et al.: Genetic studies of body mass index yield new insights for obesity biology. Nature 518, 197-206 (2015)
15.
Nead, K.T.* et al.: Contribution of common non-synonymous variants in PCSK1 to body mass index variation and risk of obesity: A systematic review and meta-analysis with evidence from up to 331 175 individuals. Hum. Mol. Genet. 24, 3582-3594 (2015)
16.
Qi, Q.* et al.: Dietary intake, FTO genetic variants and adiposity: A combined analysis of over 16,000 children and adolescents. Diabetes 64, 2467-2476 (2015)
17.
Shungin, D.* et al.: New genetic loci link adipose and insulin biology to body fat distribution. Nature 518, 187-196 (2015)
18.
Winkler, T.W.* et al.: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 11:e1005378 (2015)
19.
Arking, D.E.* et al.: Genetic association study of QT interval highlights role for calcium signaling pathways in myocardial repolarization. Nat. Genet. 46, 826-836 (2014)
20.
Hoggart, C.J.* et al.: Novel approach identifies SNPs in SLC2A10 and KCNK9 with evidence for parent-of-origin effect on Body Mass Index. PLoS Genet. 10, 1-12:e1004508 (2014)