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Eberlein, B.* et al.: Optimizing of the basophil activation test: Comparison of different basophil identification markers. Cytometry B Clin. Cytom. 88, 183-189 (2015)
Balzer, L.* et al.: Basophil activation test using recombinant allergens: Highly specific diagnostic method complementing routine tests in wasp venom allergy. PLoS ONE 9:e108619 (2014)
Cifuentes, L.* et al.: Reply. J. Allergy Clin. Immunol. 134, 494-495 (2014)
Eberlein, B.* et al.: Basophil activation test: Comparison of different basophil identification markers by multicolor flowcytometry. Allergy 69, 336 (2014)
Quaranta, M. et al.: Intraindividual genome expression analysis reveals a specific molecular signature of psoriasis and eczema. Sci. Transl. Med. 6:244ra90 (2014)
Quaranta, M. et al.: Allergic contact dermatitis in psoriasis patients: Typical, delayed, and non-interacting. PLoS ONE 9:e101814 (2014)
Quaranta, M.* et al.: Intraindividual genome expression analysis reveals opposing immune reaction in psoriasis and eczema. Br. J. Dermatol. 171, E135 (2014)
Staumont-Salle, D.* et al.: CX3CL1 (fractalkine) and its receptor CX3CR1 regulate atopic dermatitis by controlling effector T cell retention in inflamed skin. J. Exp. Med. 211, 1185-1196 (2014)
Cifuentes, L.* et al.: Identification of Hymenoptera venom-allergic patients with negative specific IgE to venom extract by using recombinant allergens. J. Allergy Clin. Immunol. 133, 909–910 (2013)
Pennino, D. et al.: IL-22 suppresses IFN-γ-mediated lung inflammation in asthmatic patients. J. Allergy Clin. Immunol. 131, 562-570 (2013)
Pennino, D.: Interleukin-17 and Interleukin-22: Key cytokines in tissue inflammation. München, Technische Universität, Fakultät für Medizin, Diss., 2013, 105 S.
Stott, B.* et al.: Human IL-31 is induced by IL-4 and promotes TH2-driven inflammation. J. Allergy Clin. Immunol. 132, 446-454 (2013)
Cavani, A.* ; Pennino, D. & Eyerich, K.: Th17 and Th22 in skin allergy. Chem. Immunol. Allergy 96, 39-44 (2012)
Eyerich, S. et al.: IL-22 and TNF-α represent a key cytokine combination for epidermal integrity during infection with Candida albicans. Eur. J. Immunol. 41, 1894-1901 (2011)
Eyerich, S. et al.: Mutual antagonism of T cells causing psoriasis and atopic eczema. N. Engl. J. Med. 365, 231-238 (2011)
Carbone, T.* et al.: CD56highCD16-CD62L- NK cells accumulate in allergic contact dermatitis and contribute to the expression of allergic responses. J. Immunol. 184, 1102-1110 (2010)
Pennino, D.* et al.: IL-17 amplifies human contact hypersensitivity by licensing hapten nonspecific Th1 cells to kill autologous keratinocytes. J. Immunol. 184, 4880-4888 (2010)
Eyerich, K.* et al.: IL-17 in atopic eczema: Linking allergen-specific adaptive and microbial-triggered innate immune response. J. Allergy Clin. Immunol. 123, 59-66.e4 (2009)
Eyerich, S.* et al.: Th22 cells represent a distinct human T cell subset involved in epidermal immunity and remodeling. J. Clin. Invest. 119, 3573-3585 (2009)