PuSH - Publication Server of Helmholtz Zentrum München

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1.
Bentley, A.R.* et al.: Multi-ancestry genome-wide gene–smoking interaction study of 387,272 individuals identifies new loci associated with serum lipids. Nat. Genet. 51, 636-648 (2019)
2.
de Vries, P.S.* et al.: Multi-ancestry Genome-Wide Association study of lipid levels incorporating gene-alcohol interactions. Am. J. Epidemiol. 188, 1033-1054 (2019)
3.
Sung, Y.J.* et al.: A multi-ancestry genome-wide study incorporating gene-smoking interactions identifies multiple new loci for pulse pressure and mean arterial pressure. Hum. Mol. Genet., accepted (2019)
4.
Evangelou, E.* et al.: Genetic analysis of over 1 million people identifies 535 new loci associated with blood pressure traits. Nat. Genet. 50, 1412-1425 (2018)
5.
Evangelou, E.* et al.: Erratum to: Genetic analysis of over 1 million people identifies 535 new loci associated with blood pressure traits (Nature Genetics, (2018), 50, 10, (1412-1425), 10.1038/s41588-018-0205-x). Nat. Genet., accepted (2018)
6.
Sung, Y.J.* et al.: A large-scale multi-ancestry genome-wide study accounting for smoking behavior identifies multiple significant loci for blood pressure. Am. J. Hum. Genet. 102, 375-400 (2018)
7.
Howson, J.M.M.* et al.: Fifteen new risk loci for coronary artery disease highlight arterial-wall-specific mechanisms. Nat. Genet. 49, 1113-1119 (2017)
8.
Wain, L.V.* et al.: Novel blood pressure locus and gene discovery using genome-wide association study and expression data sets from blood and the kidney. Hypertension 70, e4-e19 (2017)
9.
Wheeler, E.* et al.: Impact of common genetic determinants of Hemoglobin A1c on type 2 diabetes risk and diagnosis in ancestrally diverse populations: A transethnic genome-wide meta-analysis. PLoS Med. 14:e1002383 (2017)
10.
de Vries, P.S.* et al.: A meta-analysis of 120,246 individuals identifies 18 new loci for fibrinogen concentration. Hum. Mol. Genet. 25, 358-370 (2016)
11.
Ehret, G.B.* et al.: The genetics of blood pressure regulation and its target organs from association studies in 342,415 individuals. Nat. Genet. 48, 1171-1184 (2016)
12.
Golbus, J.R.* et al.: Common and rare genetic variation in CCR2, CCR5, or CX3CR1 and risk of atherosclerotic coronary heart disease and glucometabolic traits. Circ. Cardiovasc. Genet. 9, 250-258 (2016)
13.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease. N. Engl. J. Med. 374, 1134-1144 (2016)
14.
Stitziel, N.O.* et al.: Coding variation in ANGPTL4, LPL, and SVEP1 and the risk of coronary disease (vol 374, pg 1134, 2016). N. Engl. J. Med. 374, 1898-1898 (2016)
15.
Winkler, T.W.* et al.: Correction: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 12:e1006166 (2016)
16.
Interleukin 1 Genetics Consortium et al.: Cardiometabolic effects of genetic upregulation of the interleukin 1 receptor antagonist: A mendelian randomisation analysis. Lancet Diabet. Endocrinol. 3, 243-253 (2015)
17.
Holmes, M.V.* et al.: Mendelian randomization of blood lipids for coronary heart disease. Eur. Heart J. 36, 539-550 (2015)
18.
Huffman, J.E.* et al.: Modulation of genetic associations with serum urate levels by body-mass-index in humans. PLoS ONE 10:e0119752 (2015)
19.
Huffman, J.E.* et al.: Rare and low-frequency variants and their association with plasma levels of fibrinogen, FVII, FVIII, and vWF. Blood 126, e19-e29 (2015)
20.
Locke, A.E.* et al.: Genetic studies of body mass index yield new insights for obesity biology. Nature 518, 197-206 (2015)