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1.
Scott, R.A.* et al.: An expanded genome-wide association study of type 2 diabetes in Europeans. Diabetes 66, 2888-2902 (2017)
2.
Gaulton, K.J.* et al.: Genetic fine mapping and genomic annotation defines causal mechanisms at type 2 diabetes susceptibility loci. Nat. Genet. 47, 1415-1425 (2015)
3.
Albrechtsen, A.* et al.: Exome sequencing-driven discovery of coding polymorphisms associated with common metabolic phenotypes. Diabetologia 56, 298-310 (2013)
4.
Bonnefond, A.* et al.: Rare MTNR1B variants impairing melatonin receptor 1B function contribute to type 2 diabetes. Nat. Genet. 44, 297-301 (2012)
5.
Marquez, M.* et al.: Low-frequency variants in HMGA1 are not associated with type 2 diabetes risk. Diabetes 61, 524-530 (2012)
6.
Morris, A.P.* et al.: Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes. Nat. Genet. 44, 981-990 (2012)
7.
Perry, J.R.* et al.: Stratifying type 2 diabetes cases by BMI identifies genetic risk variants in LAMA1 and enrichment for risk variants in lean compared to obese cases. PLoS Genet. 8:e1002741 (2012)
8.
Dupuis, J.* et al.: New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk. Nat. Genet. 42, 105-116 (2010)
9.
Saxena, R.* et al.: Genetic variation in GIPR influences the glucose and insulin responses to an oral glucose challenge. Nat. Genet. 42, 142-148 (2010)
10.
Voight, B.F.* et al.: Twelve type 2 diabetes susceptibility loci identified through large-scale association analysis. Nat. Genet. 42, 579-589 (2010)