PuSH - Publication Server of Helmholtz Zentrum München

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1.
Prins, B.P.* et al.: Investigating the causal relationship of c-reactive protein with 32 complex somatic and psychiatric outcomes: A large-scale cross-consortium mendelian randomization study. PLoS Med. 13:e1001976 (2016)
2.
Winkler, T.W.* et al.: Correction: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 12:e1006166 (2016)
3.
Gaulton, K.J.* et al.: Genetic fine mapping and genomic annotation defines causal mechanisms at type 2 diabetes susceptibility loci. Nat. Genet. 47, 1415-1425 (2015)
4.
Segrè, A.V.* ; Wei, N.* & DIAGRAM Consortium (Florez, J.C* ; Klopp, N. ; Illig, T. ; Müller-Nurasyid, M. ; Peters, A.): Pathways targeted by antidiabetes drugs are enriched for multiple genes associated with type 2 diabetes risk. Diabetes 64, 1470-1483 (2015)
5.
Swerdlow, D.I.* et al.: HMG-coenzyme A reductase inhibition, type 2 diabetes, and bodyweight: Evidence from genetic analysis and randomised trials. Lancet 385, 351-361 (2015)
6.
Winkler, T.W.* et al.: The influence of age and sex on genetic associations with adult body size and shape: A large-scale genome-wide interaction study. PLoS Genet. 11:e1005378 (2015)
7.
Chan, Y.* et al.: An excess of risk-increasing low-frequency variants can be a signal of polygenic inheritance in complex diseases. Am. J. Hum. Genet. 94, 437-452 (2014)
8.
Claussnitzer, M. et al.: Leveraging cross-species transcription factor binding site patterns: From diabetes risk loci to disease mechanisms. Cell 156, 343-358 (2014)
9.
Gaulton, K.J.* ; Morris, A.P.* ; McCarthy, M.I.* & DIAGRAM Consortium (Gieger, C. ; Grallert, H. ; Klopp, N. ; Illig, T. ; Müller-Nurasyid, M. ; Peters, A.): FOXA2 bound sites are enriched for type 2 diabetes risk variants. Diabetologia 57, S66 (2014)
10.
DIAGRAM Consortium (Gieger, C. ; Grallert, H. ; Illig, T. ; Klopp, N. ; Müller-Nurasyid, M. ; Peters, A.) et al.: Genome-wide trans-ancestry meta-analysis provides insight into the genetic architecture of type 2 diabetes susceptibility. Nat. Genet. 46, 234-244 (2014)
11.
Hara, K.* et al.: Genome-wide association study identifies three novel loci for type 2 diabetes. Hum. Mol. Genet. 23, 239-246 (2014)
12.
Keildson, S.* et al.: Expression of phosphofructokinase in skeletal muscle is influenced by genetic variation and associated with insulin sensitivity. Diabetes 63, 1154-1165 (2014)
13.
Morris, A.P.* et al.: Fine-mapping type 2 diabetes susceptibility loci with high-density imputation. Diabetologia 57, S50-S51 (2014)
14.
Ng, M.C.Y.* et al.: Meta-analysis of genome-wide association studies in African Americans provides insights into the genetic architecture of type 2 diabetes. PLoS Genet. 10:e100451 (2014)
15.
Xi, B.* et al.: Associations of genetic variants in/near body mass index-associated genes with type 2 diabetes: A systematic meta-analysis. Clin. Endocrinol. 81, 702-710 (2014)
16.
Albrechtsen, A.* et al.: Exome sequencing-driven discovery of coding polymorphisms associated with common metabolic phenotypes. Diabetologia 56, 298-310 (2013)
17.
Köttgen, A.* et al.: Genome-wide association analyses identify 18 new loci associated with serum urate concentrations. Nat. Genet. 45, 145-154 (2013)
18.
Lam, V.K.* et al.: Genetic associations of type 2 diabetes with islet amyloid polypeptide processing and degrading pathways in asian populations. PLoS ONE 8:e62378 (2013)
19.
Li, H.* et al.: A genome-wide association study identifies GRK5 and RASGRP1 as type 2 diabetes loci in Chinese Hans. Diabetes 62, 291-298 (2013)
20.
Ma, R.C.* et al.: Genome-wide association study in a Chinese population identifies a susceptibility locus for type 2 diabetes at 7q32 near PAX4. Diabetologia 56, 1291-1305 (2013)